CHAPTER XII

APPENDICITIS, OR NATURE'S REMNANT SALE

We were not made all at once, nor do we go to pieces all at once, like

the "one-hoss shay." This is largely because we are not all of the same

age, clear through. Some parts of us are older than other parts. We have

always felt a difficulty, not to say a delicacy, in determining the age

of a given member of the human species--especially of the gentler sex.

Now we know the reason of it. From the biologic point of view, we are

not an individual, but a colony; not a monarchy, but a confederacy of

organ-states, each with its millions of cell-citizens. It is not merely

editors and crowned heads who have a biologic right to say "We."

Therefore, obviously, any statement that we make as to our age can be

only in the nature of an average struck between the ages of our heart,

lungs, liver, stomach; and as these vary in ancientness by thousands of

years, the average must be both vague and misleading. The only reason

why there is a mystery about a woman's age is that she is so intensely

human and natural. The only statement as to our age that the facts would

strictly justify us in making must partake of the vagueness of Mr. A.

Ward's famous confession that he was "between twenty-three summers."

As we individually climb our own family-tree, from the first, one-celled

droplet of animal jelly up, none of our organs is older than we are,

but a number of them are younger. The appendix is one of these. Now, by

some curious coincidence, explain it as we may, some of our oldest

organs are youngest, in the sense of most vigorous, elastic, and

resisting, while some of our youngest are oldest, in the sense of

decrepit, feeble, and unstable. It is perhaps only natural that an organ

like the stomach, for instance, which has a record of honorable service

and active duty millions of years long, should be better poised, more

reliable, and more resourceful than one which, like the lung or the

appendix, has, as it were, a "character" of only about one-tenth of that

length. However this may be, the curious fact confronts us that

scattered about through the body are structures and fragments, the

remains of organs which at one time in our ancestral career were, under

the then existing circumstances, of utility and value, but have now

become mere survivals, remnants,--in the language of the day, "back

numbers." Some of these have still a certain degree of utility, though

diminished and still diminishing in size and functional importance, like

our third molars or "wisdom" teeth, our fifth or "little" toes, our

gall-bladder, our coccyx or tail-bone, the hair-glands scattered all

over the now practically hairless surface of our bodies, and our once

movable ears, which can no longer be "pricked," or laid back. These,

though of far less utility and importance than they obviously were at

one time, still earn their salt, and, though all capable of causing us

considerable annoyance on slight provocation, seldom give rise to

serious trouble or inconvenience. There are, however, a few of these

"oversights" which are of little or no known utility, and yet which,

either by their structure or situation, may become the starting-point of

serious trouble.

The best known members of this small group are the openings through the

abdominal wall, which, originally placed at the strongest and safest

position in the quadrupedal attitude, are now, in the erect attitude, at

the weakest and most dangerous, and furnish opportunity for those

serious and sometimes fatal escapes of portions of the intestines which

we call hernia; the tonsils; and our friend the _appendix vermiformis_.

For once its name expresses it exactly. It _is_ an "appendix," an

afterthought; and it is "_vermiformis_," a worm-like creature,--and,

like the worm, will sometimes turn when trodden on. Its worm-likeness is

significant in another sense also, in that it is this very

diminutiveness in size--the coils into which it is thrown, the spongy

thickness of its walls, and the readiness with which its calibre or its

circulation is blocked--that is the fundamental cause of its tendency to

disease.

The cause of appendicitis is the appendix.

"Despise not the day of small things" is good pathology as well as

Scripture. Here we have a little, worm-shaped tag, or side branch, of

the food-tube, barely three or four inches long, of about the diameter

of a small quill and of a calibre that will barely admit an ordinary

knitting needle. And yet we speak of it with bated breath. When we

remember that this little, twisted, blind tube opens directly out of one

of the largest pouches of the intestines (the _cæcum_), and that it is

easy for anything that may be present in the large pouch--food,

irritating fragments of waste matter, or bacteria--to find its way into

this fatal little trap, but very difficult to find the way out again, we

can form some idea of what a literal death-trap it may become.

How did such a useless and dangerous structure ever come to develop in a

body in which for the most part there is mutual helpfulness, utility,

and perfect smoothness of working through all the great machine? To

attempt to answer this would carry us very far back into ancient

history. But to make such backward search is absolutely the only means

of reaching an answer.

"But," some one will object, "how perfectly irrational, not to say

absurd, to propose to go back hundreds of thousands of years into

ancient history, to account for a disease which has been

discovered--according to some, invented--within the past twenty-five

years!"

Appendicitis is a mark, not a result, of a high grade of civilization.

To have had an operation for it is one of the insignia of modern rank

and culture. Our new biologic aristocracy, the "Appendix-Free," look

down with gentle disdain upon their appendiciferous fellows who still

bear in their bodies this troublesome mark of their lowly origin. In

short, the general impression prevails that appendicitis is a new

disease, a disease which has become common, or perhaps occurred at all,

only within the last quarter of a century, and which therefore--with the

usual flying leap of popular logic--is a serious menace to our future,

if it keeps on increasing in frequency and ferocity at anything like

the same rate which it has apparently shown for the past fifteen years.

As this feeling of apprehension is in many minds quite genuine, it may

be well to say briefly, before proceeding further, first, that, if there

be any disease which absolutely and almost exclusively depends upon

definite peculiarities of structure, it is appendicitis, and that these

structural peculiarities of this tiny, cramped tag of the food-canal

have existed from the earliest infancy of the race. So it is almost

unthinkable that man should not have been subject to fatal disturbances

of this organ from the very earliest times. On the post-mortem table,

the appendix of the lowest savage is the same useless, shriveled, and

inflammable worm as that of the most highly civilized Aryan, though

perhaps an inch or so longer. Secondly, there is absolutely no adequate

proof that appendicitis is increasing in frequency among civilized

races. It is only about twenty-five years ago that it was first

definitely described, and barely fifteen that the profession began at

all generally to recognize it.

But all of us whose memory extends backward a quarter of a century can

clearly recall that, while we did not see any cases of "appendicitis,"

we saw dozens of cases of "acute enteritis," "idiopathic (self-caused)

peritonitis," "acute inflammation of the bowels," "acute obstruction of

the bowels," of which patients died both painfully and promptly, and

which we now know were really appendicitis.

In short, from a careful study of all the data, including the claims so

frequently made of freedom from appendicitis on the part of Oriental

races, colored races, less civilized tribes, vegetarians, and others, we

are tending toward the conclusion that the percentage of appendicitis in

a given community is simply the percentage of its recognition,--in other

words, of the intelligence and alertness, first of its physicians, and

then of its laity. As an illustration, my friend Dr. Bloodgood kindly

had the statistics of the surgical patients treated in the great Johns

Hopkins Hospital at Baltimore investigated for me, and found almost

precisely the same percentage of cases of appendicitis among colored

patients as among white patients.

The earlier impression, first among physicians and now in the laity,

that appendicitis is an almost invariably fatal disease, is not well

founded, and we now know that a large percentage of cases recover, at

least from the first attack; so that it is quite possible for from half

to two-thirds of the cases of appendicitis actually occurring in a given

community to escape recognition, unless promptly reported, carefully

examined, and accurately diagnosed. Thirdly, in spite of the remarkable

notoriety which the disease has attained, the general dread of its

occurrence,--which has been recently well expressed in a statement that

everybody either has had it, or expects to have it, or knows somebody

who has had it,--the actual percentage of occurrence of grave

appendicitis is small. In the United States census of 1900, which was

the first census in which it was recognized as a separate cause of

death, it was responsible for only 5000 deaths in the entire United

States for the ten years preceding, or about one death in two hundred.

This rate is corroborated by the data, now reaching into thousands, from

the post-mortem rooms of our great hospitals, which report an average of

between a half and one per cent. A disease which, in spite of the

widespread terror of it, kills only one in two hundred of those who

actually die--or about one in every ten thousand of our population--is

certainly nothing to become seriously excited over from a racial point

of view.

While appendicitis is one of the "realest" and most substantial of

diseases, and, in its serious form, highly dangerous to life, there can

be little doubt that there has come, first of all, a state of mind

almost approaching panic in regard to it; and, second, a preference for

it as a diagnosis, as so much more _distingué_ than such plebeian names

as "colic," "indigestion," "enteritis," or the plain old Saxon

"belly-ache," which has reached almost the proportions of a fad. It is

certain that nowadays physicians have almost as frequently to refuse to

operate on those who are clamoring for the distinction, as to urge a

needed operation upon those unwilling to submit to it.

The satirical proposal that a "closed season" should be established by

law for appendicitis as for game birds, during which none might be

taken, would apply almost as often to the laity as to the profession,

even the surgical half.

Since the chief cause of appendicitis is the appendix, the first

question for disposal is, How did the appendix become an appendix? To

this biology can render a fairly satisfactory answer. It is the remains

of one of Mother Nature's experiments with her 'prentice hand upon the

mammalian food-tube. As is now generally known, the food-canal in

animals was originally a comparatively straight tube, running the length

of the body from mouth to anus. It early distends into a moderate pouch,

about a third of the way down from the mouth, forming a _stomach_, or

storage and churning-place for the food. Below this, it lengthens into

coils (the so-called _small intestine_), which, as the body becomes more

complex, increase in number and length until they reach four to ten

times the length of the body. Later, the lower third of the tube

distends and sacculates out into a so-called _large intestine_, in which

the last remnants of nutritive material and of moisture are extracted

from the food-residues before they are discharged from the body. Just at

the junction of this large intestine with the small intestine, nature

took it into her head to develop a second pouch, a sort of copy of the

stomach. This pouch, from the fact that it ends in a blind sac, is known

as the _cæcum_ (or "blind" pouch), and is apparently simply a means of

delaying the passage of the foodstuffs until all the nutriment and

moisture have been absorbed out of them for the service of the body.

Naturally, it has developed to the largest degree and size in those

animals which have lived upon the bulkiest and grassiest of foods, the

so-called _Herbivora_, or grass-eaters. In the _Carnivora_, or

flesh-eaters, it is usually small, and in one family, the bears,

entirely absent. This pouch is no mere figure of speech, as may be

gathered from the fact that in certain of the rodent _Herbivora_, like

the common guinea-pig, it may have a capacity equal to all of the rest

of the alimentary canal, and in the horse it will hold something like

four times as much as the stomach. Oddly enough, among the grass-eaters,

for some reason which we do not understand, it appears to occur in a

sort of inverse proportion to the stomach; those which have large,

sacculate, pouched stomachs, like the cow, sheep, and the ruminants

generally, having smaller _cæca_. In other _Herbivora_ with small

stomachs, like the rabbit and the horse, it develops greater size.

Our primitive ancestors were mixed feeders, and, though probably more

largely herbivorous than we are to-day, had a medium-sized _cæcum_, and

maintained it up to the point at which the anthropoid apes began to

branch off from our family-tree. But at about this point, for some

reason, possibly connected with the increasing variety and improved

quality and concentration of the food, due to greater intelligence and

ability to obtain it, this large _cæcum_ became unnecessary, and began

to shrivel.

Here, however, is where nature makes her first afterthought mistake.

Instead of allowing this pouch to contract and shrivel uniformly

throughout its entire length, she allowed the farther (or _distal_)

two-thirds of it to shrivel down at a much faster rate than the central

(or _proximal_) third; so that the once evenly distended sausage-shaped

pouch, about six to eight inches long and two inches in diameter, has

become distorted down into a narrow, contracted end portion, about a

quarter of an inch in diameter, and a distended first portion, for all

the world like a corncob pipe with a crooked stem and an unusually large

bowl. And behold--the modern _appendix vermiformis_, with all its fatal

possibilities!

If we want something distinctly human to be proud of, we may take the

appendix, for man is the only animal that has this in its perfection. A

somewhat similarly shriveled last four inches of the _cæcum_ is found in

the anthropoid apes and in the wombat, a burrowing marsupial of

Australia. In some of the monkeys, and in certain rodents like the

guinea-pig, a curious imitation appendix is found, which consists simply

of a contracted last four or five inches of the _cæcum_, which, however,

on distention with air, is found to relax and expand until of the same

size as the rest of the gut.

The most strikingly and distinctly human thing about us is not our

brain, but our appendix. And, while recognizing its power for mischief,

it is only fair to remember that it is an incident and a mark of

progress, of difficulties overcome, of dangers survived. In all

probability, it was our change to a more carnivorous diet, and

consequently predatory habits, which enabled our ancestors to step out

from the ruck of the "_Bandar-Log_," the Monkey Peoples. An increase in

carnivorousness must have been a powerful help to our survival, both by

widening our range of diet, so that we could live and thrive on anything

and everything we could get our hands on, and by inspiring greater

respect in the bosoms of our enemies. Let us therefore respect the

appendix as a mark and sign of historic progress and triumph, even

while recognizing to the full its unfortunate capabilities for mischief.

But what has this ancient history to do with us in the twentieth

century? Much in every way. First, because it furnishes the physical

basis of our troubles; and second, and most important, because, like

other history, it is not merely repeating itself, but continuing. This

process of shriveling on the part of the appendix is not ancient history

at all, but exceedingly modern; indeed, it is still going on in our

bodies, unless we are over sixty-five years of age.

In the first place, we have actually passed through two-thirds of this

process in our own individual experience.

At the first appearance of the _cæcum_, or blind pouch, in our prenatal

life, it is of the same calibre as the rest of the intestine, and of

uniform size from base to tip. About three weeks later the tip begins to

shrivel, and from this on the process steadily continues, until at birth

it has contracted to about one-fifteenth of the bulk of the _cæcum_. But

the process doesn't stop here, though its progress is slower. By about

the fifth year of life the stem of the cæco-appendix pipe has diminished

to about one-thirtieth of the size of the bowl, which is the proportion

that it maintains practically throughout the rest of adult life. For a

long time we concluded that the process was here finished, and that the

appendix underwent no further spontaneous changes during life; but,

after appendicitis became clearly recognized, a more careful study was

made of the condition of the appendix in bodies coming to the

post-mortem table, dead of other diseases, at all ages of life. This

quickly revealed an extraordinary and most significant fact, that, while

the appendix was no longer decreasing in apparent size, its internal

capacity or calibre was still diminishing, and at such a rate that by

the thirty-fifth year it had contracted down so as to become cut off

from the cavity of the _cæcum_ in about twenty-five to thirty per cent

of all individuals. By the forty-fifth year, according to the anatomist

Ribbert (who has made the most extensive study of the subject), nearly

fifty per cent of all appendices are found to be cut off, and by the

sixty-fifth year nearly seventy per cent.

This explains at once why appendicitis is so emphatically a disease of

young life, the largest number of cases occurring before the

twenty-fifth year (fifty per cent of all cases occur between ten and

thirty years of age), and becoming distinctly rarer after the

thirty-fifth, only about twenty per cent occurring after this age. As

soon as the cavity of the appendix is cut off from that of the

intestine, it is of course obvious that infectious or other irritating

materials can no longer enter its cavity to cause trouble, although, of

course, it is still subject to accidents due to kinks, or twists, or

interference with its blood-supply; but these are not so dangerous,

providing there be no infectious germs present.

Here, then, we have a clear and adequate physical basis for

appendicitis. A small, twisted, shriveling spur or side twig of the

intestine, opening from a point which has become a kind of settling

basin in the food-tube, its mouth gaping, as it were, to admit any

poisonous or irritating food, infectious materials, disease-germs, the

ordinary bacteria which swarm in the alimentary canal, or irritating

foreign bodies, like particles of dirt, sand, hairs, fragments of bone,

pins, etc., which may have been accidentally swallowed. Once these

irritating and infectious materials have entered it, spasm of its

muscular coat is promptly set up, their escape is blocked, and a violent

inflammation easily follows, which may end in rupture, perforation, or

gangrene.

Not only may any infection which is sweeping along the alimentary canal,

thrown off and resisted by the vigorous, full-sized, well-fed intestine,

find a point of lowered resistance and an easy victim for its attack in

the appendix, but there is now much evidence to indicate that the

ordinary bacteria which inhabit the alimentary canal, particularly that

first cousin of the typhoid bacillus, the colon bacillus, when once

trapped in this _cul-de-sac_, may quickly acquire dangerous powers and

set up an acute inflammation. It is not necessary to suppose that any

particular germ or infection causes appendicitis. Any one which passes

through, or attacks, the alimentary canal is quite capable of it, and

probably does cause its share of the attacks.

Numerous attempts have been made to show that appendicitis is

particularly likely to follow typhoid fever, rheumatism, influenza,

tonsilitis, and half a dozen other infectious or inflammatory processes.

But about all that has been demonstrated is that it may follow any of

them, though in none with sufficient frequency or constancy to enable it

to be regarded as one of the chief or even one of the important causes

of the disease.

One dread, however, we may relieve our anxious souls of, and that is the

famous grape-seed or cherry-stone terror. To use a Hibernianism, one of

our most positive conclusions in regard to the cause of appendicitis is

a negative one: that it is not chiefly, or indeed frequently, due to the

presence of foreign bodies. This was a most natural conclusion in the

early days of the disease, since, given a tiny blind pouch with a

constricted opening gaping upon the cavity of the food-canal, nothing

could be more natural than to suppose that small irritating food

remnants or foreign bodies, slipping into it and becoming lodged, would

block it and give rise to serious inflammation. And, moreover, this _a

priori_ expectation was apparently confirmed by the discovery, in many

appendices removed by operation, of small oval or rounded masses,

closely resembling the seed of some vegetable or fruit. Whereupon

anxious mothers promptly proceeded to order their children to "spit

out," with even more religious care than formerly, every grape-seed and

cherry-stone. The increased use of fresh and preserved fruits was

actually gravely cited, particularly by our Continental brethren, as one

of the causes of this new American disease. Barely ten years ago I was

spending the summer in the Adirondacks, and was bitterly reproached by

the host of one of the Lake hotels, because the profession had so

terrified the public about the dangers of appendicitis from fruit-seeds

that he was utterly unable to serve upon his tables a large stock of

delicious preserved and canned raspberries, blackberries, and grapes

which he had put up the previous years. "Why," he said, "more than half

the people that come up here will no more eat them than they would

poison, for fear that some of the seeds will give 'em appendicitis."

This dread, however, has been deprived of all rational basis, first, by

finding that many inflamed appendices removed, after the operation

became more common, contained no foreign body whatever; secondly, that

many perfectly healthy appendices examined on the post-mortem table,

death being due to other diseases, contain these apparently foreign

bodies; and thirdly, that when these "foreign bodies" were cut into,

they were found to be not seeds or pits of any description, but hardened

and, in some cases, partially calcareous masses of the fæces.

We are in a nearly similar position in regard to the third alleged cause

of appendicitis, and that is food. Many are the accusations which have

been made in this field. On the one hand, meat and animal foods

generally have been denounced, on account of their supposed "heating" or

"uric-acid-forming" properties; while on the other, vegetables and

fruits have been equally hotly incriminated, on account of their seeds,

fibres, husks, and irritating substances, and the danger of their being

contaminated by bacteria and other parasites from the soil. These

charges appear to have little adequate foundation, and, so far as we are

in a position now to judge, the only way a food can give, or be

accessory to, appendicitis is by its being taken in such excessive

amounts as to set up fermentive or putrefactive changes in the

alimentary canal, or by its being in an unsound, decaying, or actually

diseased condition. Any amounts or quality of food which are capable of

giving rise to an attack of acute indigestion may secondarily lead to an

attack of appendicitis. The only single article of diet whose ingestion

is declared by Osler to be rather frequently followed by an attack of

appendicitis is the peanut.

Therefore, the best thing to do in the way of taking precautions against

the occurrence of appendicitis is, in the language of the day, to

"forget it" as completely as possible, reassuring ourselves that, in

spite of its extraordinary notoriety and popularity, it is a

comparatively rare disease in its fatal form, responsible for not more

than one-half of one per cent of the deaths, and that the older we grow,

the better become our chances of escaping it.

Whatever we may have decided in regard to our brains, by the time we

reach fifty, we may feel reasonably sure we've no appendix.

But the question will at once arise, if the appendix be so tiny in size,

so insignificant in capacity, and so devoid of useful function, what is

the use of disturbing ourselves over the question of what may become of

it? If it is going to decay and drop off, why not permit it to do so,

with the philosophic indifference with which we would sacrifice the tip

of our little fingers in a planing-mill? Here, however, is just the rub,

and the fact that gives to appendicitis all its terrors, and to the

question of what to do in each particular case its difficulties and

perplexities.

The appendix does not, unfortunately, hang out from the surface of the

body, where it could peacefully decay and drop off without prejudice to

the rest of the body, or be quickly lopped off in the event of its

giving trouble. On the contrary, it projects its stubby and

insignificant length right into the midst of the most delicate and

susceptible cavity of the body, the general cavity of the abdomen, or

peritoneum. The thin, sensitive sheet of peritoneum which lines this

cavity covers every fold and part of the food-tube, from the stomach

down to the rectum. And when once infection or inflammation has occurred

at any point in it, there is nothing to prevent its spreading like a

prairie fire, all over the entire abdominal cavity from diaphragm to

pelvis. If this wretched little remnant were a coil of explosive fuse

within the brain-cavity itself, which any jar might set off, it could

hardly be richer in possibilities of danger.

A redeeming feature of appendicitis is that the appendix lies--so to

speak--in a corner, or wide-mouthed pouch, of the great peritoneal

cavity; and if the inflammation set up in it can be "walled off" from

the rest of the peritoneal cavity, and limited strictly to this little

corner or pouch, all will be well. This is what occurs in those cases of

severe appendicitis which spontaneously recover. If, however, this

disturbance bursts its barriers, and lights up an inflammation of the

entire peritoneal cavity, then the result is likely to be a fatal one.

Just how far nature can be trusted in each particular case to limit and

stamp out the process in this manner is the core of the problem that

confronts us, as attending physicians.

In the majority of cases, fortunately, the peritoneal fire brigade acts

promptly, pours out a wall of exudate, and locks up the appendix in a

living prison, to fight out its own battles and sink or swim by itself.

But unfortunately, in a minority of cases, by a wretched sort of

"senatorial courtesy" which exists in the body, the appendix is given

its ancestral or traditional rights and allowed to inflict its petty

troubles upon the entire abdominal cavity, and include the body in its

downfall.

Lastly come the two most pertinent and appealing questions:--

What is the outlook for me if I should develop appendicitis? And what is

to be done?

In regard to the first of these, it is safe to say that our answer is

much less alarming than it was in the earlier stage of our knowledge.

Naturally enough, in the beginning, only the severest and most

unmistakable forms of the disease and those which showed no tendency to

localization, were recognized, or at least came under the eye of the

surgeon; and as a large percentage of these resulted fatally, the

conclusion was reached, both in the medical profession and by the laity,

that appendicitis was an exceedingly dangerous disease, with a high

fatality in all cases. As, however, physicians became more expert in the

recognition of the disease, it was discovered to be vastly more common,

while side by side came the consoling knowledge that a considerable

percentage of cases got well of themselves, in the sense of the

inflammation being limited to the lower right-hand corner of the

abdominal cavity, though, of course, with the possibility of leaving a

smouldering fuse which might light up another explosion under any stress

in future.

Further, as the attention of the post-mortem investigators at our large

hospitals was directed to the subject, it was found that a very

considerable percentage of all bodies, ranging from twenty to--according

to some estimates--as high as sixty per cent, showed changes in the

appendix and its neighborhood which were believed to be due to old

inflammations; so that, while it is possible to speak only with great

caution and reserve, the balance of opinion among clinicians and

pathologists of wide experience and the more conservative surgeons

appears to be that from one-half to two-thirds of all cases of

appendicitis will recover of themselves, in the sense of subsiding more

or less permanently, without causing death.

On the other hand, it must be remembered that the appendix is an organ

which, so far as any evidence has been adduced, is entirely without

useful function; that it is in process of shriveling and disappearance,

if left entirely alone, and that the best result which can be expected

from a self-cured attack of appendicitis is the destruction of the

appendix and its elimination as a further possible cause of mischief. By

avoiding an operation in appendicitis, we may be practically certain

that we save nothing that is worth saving--except the fee. Moreover,

even though only from one-fourth to one-third of all cases develop

serious complications, you never can be quite sure in which division

your particular case will fall.

The situation is in fact a little bit like one related in the

experience of Edison, the inventor. The trustees of a church in a

neighboring town had just completed a beautiful new church building with

a high spire, projecting far above any other building in the town. When

it was nearing completion, the question arose, should they put on a

lightning-rod. The great church itself had strained their financial

resources, and one party in the board were of the opinion that they

should avoid this unnecessary expense, supporting their economic

attitude by the argument that, to put on a lightning-rod, would argue a

lack of trust in Providence. Finally, after much debate, it was decided,

as the great electrician was readily accessible, to submit the question

to him. Mr. Edison listened gravely to the arguments presented, pro and

con.

"What is the height of the building, gentlemen?"

The number of feet was given.

"How much is that above that of any surrounding structures?"

The data were supplied.

"It is a church, you say?"

"Yes."

"Well," said the great man, "on the whole, I should advise you to put on

a lightning-rod. Providence is apt to be, at times, a trifle

absent-minded."

The chances are in favor of your recovery, but--put on a lightning-rod,

in the shape of the best and most competent doctor you know, and be

guided entirely by his opinion. An attack of appendicitis is like

shooting the Grand Lachine Rapids. Probably you will come through all

right; but there is always the possibility of landing at a moment's

notice on the rocks or in the whirlpools. With a good pilot your risk

doesn't exceed a fraction of one per cent. And fortunately this

condition has been not merely theoretically but practically reached

already; for the later series of case-groups of appendicitis treated in

this intelligent way by coöperation between the physician and surgeon

from the start, with prompt interference in those cases which to the

practiced eye show signs of making trouble, has reduced the actual

recorded mortality of the disease to between two and five per cent. Even

of those cases which come to operation now, the death-rate has been

reduced as low as five per cent, in series of from 400 to 600 successive

operations. When we contrast this with the first results of operation,

when the cases as a rule were seen too late for the best time of

interference, and from twenty per cent to thirty per cent died; and with

the intermediate stage, when surgeons as a rule were inclined to advise

operation at the earliest possible moment that the disease could be

recognized, and from ten per cent to fifteen per cent died, we can see

how steady the improvement has been, and how encouraging the outlook is

for the future.

Cases which have weathered one attack of appendicitis are of course by

no means free from the risk of another. Indeed, at one time it was

believed that a recurrence was almost certain to occur. Later

investigations, based upon larger numbers of cases, now running up into

the thousands, give the reassuring result that though this danger is a

real one, it is not so great as it was at one time supposed, as the

average number in whom a second attack occurs appears to be about

twenty per cent. This, however, is a large enough risk to be worthy of

serious consideration; and in view of the fact that the mortality of

operations done between attacks is less than one per cent, it is

generally the feeling of the profession that, where there is any

appreciable soreness, or tenderness, or liability to attacks of pain in

the right iliac region, in an individual who has had one attack of

appendicitis, the really conservative and prudent procedure is to have

the source of the trouble removed once and for all.

The four principal symptoms of appendicitis are: pain, which is usually

felt most keenly somewhere between the umbilicus and the right groin,

though this is by no means invariable; tenderness in that same region

upon pressure; rigidity of the muscles of the abdominal wall on the

right side; and temperature, or fever.

No matter how much and how variegated pain you may have in the abdomen,

or how high your temperature may run, if you are not distinctly sore on

firm pressure down in this right lower or southwest quadrant of the

abdomen,--but be careful not to press too hard, it isn't safe,--you may

feel fairly sure that you haven't got appendicitis. If you are, you may

still not have it, but you'd better send for the doctor, to be sure.

Preventable Diseases · 第 12 / 19 章