Bulbar Paralysis

(LABIOGLOSSOLARYNGEAL PARALYSIS)

A progressive atrophy and paralysis, invading the lips, tongue, pharynx

and larynx, due to involvement (sclerosis) of the motor nuclei of the

medulla oblongata that supply these tissues. It is rarely primary,

more =frequently secondary= to tabes, amyotrophic lateral sclerosis

and diseases involving the motor nuclei of the medulla. Diphtheria,

syphilis and lead poisoning are said to predispose. =Osteopathic

lesions= of the upper cervical are also important factors in many

cases. Halbert says: “The nuclei of the hypoglossal, the spinal

accessory, the facial and the motor part of the trifacial nerves suffer

most decidedly from the sclerotic degeneration. The nerve trunks and

the muscles which they supply gradually show the effects of a similar

degeneration.”

The =acute form= results from hemorrhage, embolism or inflammatory

softening. The onset is usually sudden. The speech is difficult or

entirely lost. There are dribbling of saliva, difficult swallowing,

flabbiness and flaccidity of the lips and frequent choking spells

occur. These cases may prove rapidly fatal.

The =chronic form= may result from progressive muscular atrophy,

insular sclerosis, amyotrophic lateral sclerosis, acute ascending

paralysis or chronic poliomyelitis. The paralysis starts in the tongue,

the first symptom being a slight defect in the speech. When the lips

become involved, the patient cannot whistle and speech is rendered

still more difficult. The lips are prominent and the lower one drops.

The saliva is increased in amount and there is drooling. Mastication

of the food becomes difficult. The tongue becomes atrophied and the

mucous membrane wrinkled. Fibrillary tremors of the lips and tongue are

present. Sensory symptoms are not present. Taste is normal. Paralysis

of the larynx is not so pronounced as of the other parts.

=Diagnosis.=—This is generally easy as the symptoms are well marked.

The =prognosis= is unfavorable.

=Treatment.=—Little can be done in the majority of cases. Only in those

cases where the paralysis is caused by =cervical lesions= can much hope

be given. Derangements of the cervical vertebræ, especially the atlas

and axis, occasionally influence the circulation in the medulla to such

an extent that the motor nuclei are greatly involved. The subluxated

vertebras may interfere with the blood-vessels directly or through

the vasomotor and trophic nerves. When the onset is not abrupt, the

prognosis is more favorable. When deglutition is impaired, the stomach

tube should be used in feeding the patient to prevent the food passing

into the trachea.

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