Acute Lobar Pneumonia

(CROUPOUS PNEUMONIA)

This is an acute, infectious disease wherein various vertebral, rib

and muscular lesions predispose to a lowered nutritive state of the

parenchyma of the lung, permitting the invasion of the diplococcus

pneumoniæ, with consequent local inflammation and pronounced

constitutional disturbances, chill, extreme prostration and fever,

which terminates abruptly by crisis. Secondary infective processes are

frequent.

In describing a typical case of pneumonia it is considered as a

self-limiting disease. By osteopathic treatment it is often aborted or,

at least, its course much shortened. In such a case it is not typical

pneumonia and could not be described as such.

=Osteopathic Etiology= and =Pathology=.—Pneumonia occurs more often in

the young up to the sixth year and in the aged. It is more frequent

during the winter and spring months. “Colds,” exposure and wetting are

predisposing influences that lower resistance. Climate exerts little

predisposing influence. Males are, on the whole, more frequently

attacked. Pneumonia may follow injuries of the chest. Various

derangements of the ribs and vertebræ are always found in pneumonia;

such derangements correspond with the regions of vasomotor, motor and

trophic fibers of the lungs, viz., second to seventh dorsal, inclusive,

and the upper cervical vertebræ, the latter region affecting the vagi.

The muscles of the chest region are always severed contracted. These

various disorders produce a lowered vitality of the bronchial and lung

tissues, thus favoring the existence of the micrococcus lanceolatus.

Unhygienic surroundings, alcoholism, any or all habits that tend to

depress the nervous system, or lowered vitality from some pre-existent

disease, like diabetes, Bright’s disease, organic heart affection

or one of the infectious fevers, favor its development. One attack

undoubtedly predisposes to another and repeated attacks may occur in

the same individual. The exciting cause is the invasion of the lung by

pathogenic bacteria, especially by diplococcus pneumoniæ. Pneumococci

are frequently found in the throat and mouth of the healthy.

=Pathologically=, the lung in croupous pneumonia exhibits three

distinct stages—congestion, red hepatization and gray hepatization. In

the =stage= of =engorgement= the tissue is red in color, firm and solid

and less crepitant than the healthy lung. The cut surface is bathed in

blood and stained serum. Microscopic examination shows the capillaries

to be dilated and tortuous. The alveolar epithelium is swollen and the

air cells filled with a variable number of red corpuscles, detached

alveolar cells and a few leucocytes. During the =stage= of =red

hepatization= the tissue is solid. It is reddish brown in color and of

a dry, mottled appearance. It is very friable and does not crepitate,

as the affected portion is airless. Its weight and specific gravity

are increased so that it sinks in water. The torn surface presents a

granular appearance, there being fibrinous plugs in the air cells. On

microscopic examination the air spaces are found filled with coagulated

fibrin. The tissue contains red blood-corpuscles and pus cells and the

walls of the air cells are infiltrated. In sections properly treated

the diplococcus is detected, and in some cases also the streptococcus

and staphylococcus. In the =stage= of =gray hepatization=, the lung is

still dense and heavy, but the surface is moister and softer, while the

lung tissue is even more friable and the red color gives place to a

mottled gray. The exudate loses its granular character and a yellowish

white purulent liquid flows from a cut surface. Microscopically, the

air cells are filled with leucocytes, while the red corpuscles and

fibrin filaments have disappeared. The stage of gray hepatization is

the stage of beginning =resolution=. The exudate is softened. The cell

elements are disintegrated and absorbed by the lymphatics and largely

eliminated through the kidneys. In unfavorable cases the consolidated

lung may become infiltrated with pus, and abscesses occur. In some

instances the tissue is gangrenous, or it may become the seat of

fibroid induration. These, however, are rare.

=Symptoms.=—The disease begins abruptly, usually with a severe chill,

lasting from half an hour to an hour, the fever rising rapidly. There

is a sharp pain in the side, the skin becomes harsh and dry, the

face is flushed, the eyes are bright and the expression anxious. A

short, dry, painful cough soon develops. The expectoration presents

a characteristic, rusty or blood tinged appearance and is extremely

tenacious. The temperature rises rapidly, frequently to 104 or 105

degrees F., and continues high for from five to ten days and generally

terminates by crisis. The pulse is full, but the pulse-respiration

ratio is not maintained. There is marked dyspnea, the respirations

ranging from forty to fifty per minute. There are many fine rales.

Headache, gastro-intestinal disturbances, sleeplessness, epistaxis,

rarely delirium except in drunkards, may also be present.

The symptoms given are those of a typical case of pneumonia, but all

are subject to modification. The onset may be gradual and the chill

absent. In all cases, and especially drunkards, the temperature may not

be high, while the pulse is often feeble and rapid instead of full and

strong, and the physical signs may not make their appearance until the

second or third day.

=Special Symptoms.=—The =fever= rises abruptly in the initial chill,

the temperature reaching 104 or 105 degrees F., and is continuous with

a variation of a degree or two. The fever terminates by =crisis= after

having continued from five to nine days. The temperature commonly

falls during the night and is accompanied by a profuse perspiration.

The temperature may fall from five to eight degrees in eight to twelve

hours. There is a wide range here depending upon promptness and

skillfulness of treatment, the reaction of the tissues, and previous

health. Early treatment is invaluable in modifying the course of the

disease.

The =sputum= at first is mucoid and frothy. About the second day it

becomes of a characteristic color, quite copious and consisting of

a frothy, fluid mucus, containing small viscid masses. It is very

viscid and glutinous, in some cases almost from the onset. In old

and previously weak persons, there may be no expectoration. Under

the microscope the sputum is seen to contain red blood-corpuscles,

leucocytes, alveolar epithelium, the micrococcus lanceolatus as well

as other micro-organisms, pus corpuscles and small fibrinous casts.

A stabbing =pain= is a common early symptom, as well as a dry, short

=cough=. The =urine= is febrile, scanty and high colored. Urea and

uric acid are increased. A trace of albumin is often present, and

there may be symptoms of acute nephritis. =Herpes= is common. The

nasolabial herpes appear from the second to the fifth day, and they

may occur upon the cheek, genitals and also upon mucosa of the tongue.

It is supposed to indicate a favorable prognosis. There is redness of

the cheek, usually on the affected side. The mucous membrane of the

mouth is dry. The tongue is white and furred. Anorexia and thirst are

present. The patient is usually constipated, but diarrhea may occur.

Vomiting is common. The spleen is usually enlarged, but the liver is

not perceptibly increased in size, unless there is extreme engorgement

of the right heart. The =pulse= is bounding. The average pulse-rate

is from 100 to 108 per minute. In consolidation the left ventricle

receives a lessened amount of blood and the pulse may become small. In

the aged and debilitated, a small, weak and rapid pulse may be present.

The =heart sounds= are loud and clear, and in favorable cases the

pulmonary second sound is accentuated, owing to the increased tension

in the pulmonary vessels. Upon distension of the right side of the

heart and partial failure of the right ventricle, the second sound

becomes less distinct which is a very unfavorable symptom, for very

much depends upon the strength of the right ventricle in pneumonia.

The =blood= usually exhibits leucocytosis which disappears with the

crisis. In malignant pneumonia this is absent and its continued absence

is an unfavorable sign. The proportion of fibrin is also greatly

increased. The diplococci can rarely be seen. Headache is common as an

initial symptom and may be persistent. The disease is often ushered

in by convulsions, especially in children; consciousness is usually

retained throughout the whole attack, even in severe cases, though in

some cases there is delirium. In drunkards delirium tremens may be

present from the onset. In these cases the patient often wanders about

until the preliminary excitement gives way to coma.

=Physical Signs.=—=Stage= of =Congestion.=—Diminished expansion, the

movements of the affected side are defective, the face is flushed and

the patient lies on the affected side. Tactile fremitus is slightly

increased. There may be tympany over the involved area from diminished

intrapulmonary tension. In the latter part of this stage there is

impairment of resonance. Fine crepitant rales are heard at the end of

forced inspiration. Great care has to be taken in examination when

there is deep seated consolidation.

=Stage= of =Red Hepatization=.—The breathing is markedly abnormal. Very

little or no expansive motion of the chest over the affected region.

Vocal fremitus is markedly exaggerated. The skin is hot and dry and

the pulse frequent. Dullness over the affected parts with an increased

sense of resistance is present. There is high-pitched, prolonged,

bronchial breathing when the lung becomes solidified. When the larger

bronchi are completely filled with exudate, tubular breathing is

absent. Crepitant rales may also be heard.

=Stage= of =Gray Hepatization=.—Largely the same physical signs

are repeated in this stage as in the second. The normal manner of

breathing returns, as does also the normal expansive movement of the

affected side. Crepitant rales reappear. The temperature of the skin is

lessened, breathing changes from bronchial to vesicular and bronchial

resonance continues for some time.

=Complications.=—=Pleurisy= is the most frequent complication.

Pneumonia on one side and pleurisy on the other is possible. The pain

is more acute and localized. The respiration is greatly affected and

the usual signs of effusion are present. Empyema may be a complication.

=Pericarditis= is more common in the pneumonia of children. Though

usually plastic it may be serofibrinous, but rarely the fluid is

purulent. There is increased dyspnea, the pulse becomes weaker,

and the heart sounds are gradually suppressed. =Endocarditis= is

a comparatively frequent complication. It is more liable to attack

patients with old valvular disease and to affect the left heart. The

physical signs are sometimes absent and even when present are liable

to be very deceptive. It may, however, be suspected in cases where the

fever is protracted; when septic manifestations, such as chills, sweats

or irregular temperature, develop; when embolic symptoms appear, or

when a rough, diastolic murmur develops. =Meningitis= is a complication

that comes on at the height of the fever. This complication is

rarely recognized unless the basilar meninges are involved. It is

frequently associated with ulcerated endocarditis. Cerebral embolism

causing hemiplegia has been observed. Other possible complications

are neuritis, arthritis, nephritis, parotitis and various digestive

disorders.

=Diagnosis.=—A typical case of pneumonia is easily recognized. The

abrupt onset with rigor, the rapidly developed fever, the sputum,

physical signs and abnormal pulse-respiration ratio, as a rule make

the diagnosis easy. Frequent examination of the lungs should be made

in Bright’s disease, diabetes, organic affections of the heart,

cancer and alcoholism, as all these affections are liable to become

complicated with acute pneumonia. =Pleurisy= is often confounded with

pneumonia. The resemblance between friction sounds and crepitant rales

is often very close. In pleurisy vocal resonance and vocal fremitus are

diminished; there is no “rusty” sputum; the percussion dullness may

change with the posture of the patient, and the breathing is distant

and weak. A typhoid state may be mistaken for typhoid fever. Hypostasis

occurs late in typhoid fever while dullness sets in early in pneumonia.

The history of the onset will be of aid, as pneumonia as a complication

sets in late in the disease. The Widal test will be of value. =Acute

phthisis= may begin with a chill and may resemble pneumonia very

closely, especially the physical signs. Examination of the sputum will

show the bacilli of tuberculosis. The X-ray will often be of aid as a

diagnostic measure.

=Prognosis.=—This largely depends upon the previous health of

the patient. At the extremes of life the prognosis is much more

unfavorable. It is especially fatal in drunkards. By competent

osteopathic treatment the mortality rate may be materially lessened and

this disease, dreaded by both physician and patient, need not seem so

fearful. The death rate from pneumonia during the past few years has

been appalling. In New York and Chicago nearly one-eighth of the deaths

the year around are due to pneumonia, and during certain months of the

year twenty-seven or eight per cent. of all deaths are due to this

disease. Drug medication is notoriously unreliable, the most competent

physicians freely admitting that they are practically powerless to stay

the ravages. Given a patient with a fair constitution, osteopathic

treatment will offer reasonable hope to the sufferer. There is no

question that osteopathy merits much commendation in the treatment

of pneumonia. Many severe cases have been cured and many more have

undoubtedly been aborted. The treatment is directly applicable and

specifically indicated, and coupled with good nursing and hygiene, the

mortality rate of the old schools is being markedly lessened.

=Treatment.=—The treatment of pneumonia must be both constitutional and

local. By this is meant that the systemic strength and vigor must be

maintained in addition to treatment of the chief lesion of the disease,

which is located in the lungs.

During the various stages of the disease, the treatment should

be directed to the nerves of direct innervation that control the

capillaries, and to the vasomotor nerves of the pulmonary circulation,

in order that the hyperemic and inflamed state of the pulmonary

capillaries and adjacent tissues may be lessened and the circulatory

system equalized. The disordered tissues that should be corrected

in order that the centers of the spinal cord and the nerves that

influence the function and structure of the lungs may be relieved, are:

contraction of the thoracic and dorsal muscles, subluxations of the

ribs and dorsal vertebræ from the second to the seventh, inclusive,

and the upper cervical vertebræ that may become disordered and impinge

upon the vagi nerves. However, owing to the fact that the vasomotors

are not especially abundant here, all increased chest mobility and deep

breathing and abdominal aid will materially assist the circulation.

Also, carefully treat the middle and inferior cervical regions for the

lymphatics of the lungs. Each of these regions should be carefully

examined and thoroughly treated whenever found involved. The specific

micro-organisms that influence the course of pneumonia are naturally

very important factors; but observing and improving the general health,

and establishing an unobstructed circulation through the diseased

lung tissues will hasten the crisis by favoring a rapid formation of

antidotal substances to neutralize the poisonous substance produced by

the micrococcus lanceolatus. Healthy tissues, which occur only where

there is uninterrupted freedom of vascular supply and nerve force,

are obtained by correction of any and all anatomical disorders. This

will rapidly decrease any lethal tendency in the patient and often

abort the disorder so that all that is needed is sufficient time

for nature to heal the diseased tissues. The principal predisposing

cause of many specific diseases, is some disorder of the anatomical

tissues that interferes with normal physiological functions; and the

determining of the different types of disease is often due to the

location of the lesion and the character of the micro-organism involved

in each disease. What is necessary in many cases is a correction of

the mechanical predisposing condition and the exciting and determining

influences will be rendered inactive.

The importance of close attention to both vagi can not be

overestimated. Any obstruction above or below the origin of the

superior laryngeal nerve is followed by loss of motor power of the

lungs, thus causing difficult and labored breathing. The lungs become

surcharged with blood, because the air pressure in the lungs is low

and the thorax is distended. This condition is followed by serous

exudation. Thus obstruction of the vagi may be one factor in the cause

of pneumonia. Obstruction of the vagi below the origin of the recurrent

laryngeal nerves affects the lower and middle lobes of the lungs,

and produces also a catarrhal inflammation of the upper lobes. The

recurrent laryngeal nerves may be obstructed by dilatation of the aorta

or subclavian artery as they wind about them; also by dislocations

of the first and second ribs, which may affect the nerves not only

directly, but by causing an obstruction to the subclavian vessels with

a consequent disturbance of the aorta and the heart. The recurrent

laryngeal nerves may be treated directly at the inner lower part of the

sternomastoid.

One of the chief objects of the treatment should be to prevent =heart

failure= and to lessen the pulse-respiration ratio. The average

pulse-rate in typical cases is from 100 to 110 per minute and when

it exceeds this to any extent, say 120, there is cause for alarm. At

first the pulse is full and bounding, later it is small on account of

a lessened amount of blood reaching the left ventricle and systemic

circulation, owing to the extensive consolidation. In treating heart

failure particular attention should be paid to the condition of the

ribs on the left side over the region of the heart, the second to the

fifth, inclusive. A correction of any disturbance to the inhibitory

nerves of the heart, (the vagi) and the accelerator fibers of the heart

(the cervical sympathetic) should be made. This means close attention

to probable derangements of the vertebræ from atlas to first dorsal.

General treatment of the entire system will relieve the heart of

some work and favor an equalization of the vascular system. Also by

the use of hydrotherapy the maintenance of the heart’s action may be

accomplished. Cold compresses, and not warm ones, should be used, as

the latter relax the vessel walls, producing more or less paresis of

the vessels, while the former stimulate the vaso-dilators, producing

dilatation and tone of the vessels, thereby causing a vigorous increase

in the flow of blood. This relieves the heart by increasing the

cutaneous circulation, besides increasing arterial tension. The right

heart is indirectly aided by the increase of the tension in the general

vascular system, and the vessels of the pulmonary circulation have more

force expended upon them and a greater contraction of their vessels

occurs on account of the dilatation of the cutaneous vessels. The

temperature of the water used should be 60 degrees F., and the compress

applied for thirty minutes or as long as necessary.

Attention to the abdominal area and diaphragm will have a definite

effect upon the circulation and elimination. It is beneficial in its

influence upon lungs and heart and in combatting toxemia. Carefully

graduated deep breathing is of distinct benefit.

In addition to the fever treatment in the cervical and dorsal regions,

the gradually cooled tub-bath will be of aid. The temperature at first

should be ninety degrees F. and then gradually cooled to eighty degrees

F. The duration should not be over ten or fifteen minutes. Care should

be taken that the patient does not exert himself. He should be lifted

in and out of the baths. These baths also have a marked effect upon the

respiratory and nervous centers. The ice-bag over the chest and spine

has a beneficial influence; still, with feeble children be exceedingly

careful when applying or using cold methods.

=During all stages of the disease=, the best possible care should be

taken of the patient. See the patient frequently, probably twice a day

or oftener. Each time thoroughly relax the dorsal muscles and readjust

the ribs, for as every osteopath of experience will note (and Dr.

Still particularly emphasizes) the contracted muscles frequently and

continually displace the ribs. The treatment should not be prolonged to

a point of overfatigue, but a definite reaction of tissues should be

secured but no further.

Carefully raise all the ribs and moderately hyperextend the spine.

Release the cervical, pectoral and axillary lymphatics, and stimulate

spleen and liver.

Experience has shown that the first treatment is of the greatest

importance and if the osteopath will control the predominant symptoms

at that time the result will be much simplified. For that reason it is

best not to leave the patient until the chest pain, fever, high pulse

or whatever may be present, are well in hand, although it may mean

a long visit with fairly frequent treatments. Treat the conditions

existing and wait; then treat again and the result will more than

repay. There is always more than a chance of aborting the disease, but

the first treatment is often the crucial test. F. E. Moore and many

others report numerous cases treated without a fatality and the average

duration of the disease not exceeding five days. The apartment should

be well aired and a temperature of 65 degrees F. maintained. In the

very young the temperature should be higher. The diet is exceedingly

important. Give a liquid, light and nutritious one, a milk diet being

preferable. Otherwise give meat juice, broths, egg albumin and whey.

Avoid starchy and saccharine foods, and give plenty of water. Good

nursing and complete rest of body and mind, with careful attention to

the activity of the bowels, kidneys and skin, will indirectly aid the

clogged up lung fascia to perform its function and hasten an early

recovery from the disease. In epidemic forms be particularly vigilant

in the employment of antiseptics.

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