Acute Gastritis

Acute dyspepsia is one of the frequent disorders of the stomach. It

may occur as an early symptom of an infectious disease, but very often

it is due to some non-specific irritation. The usual exciting causes

are errors of diet, over-indulgence in improperly cooked and highly

seasoned food, or food that has been spoiled, such as meat, fish and

milk, or over or under ripe fruit. Food that is either too hot or too

cold may develop an attack. Alcohol is a common cause in those not

accustomed to its use. Overuse of tobacco may bring on an attack. Many

acute “bilious” attacks are brought about by some mental shock or

excitement at the time of taking food, for it has been shown by the

researches of Pawlow that both gastric motion and secretion are altered

by mental irritation during digestion.

Unquestionably osteopathic lesions of the splanchnics and vagi are

important predisposing factors. These lesions produce a lowered

resistance of the tissues, which will frequently explain why certain

exciting factors that will initiate an attack in one individual will

not do so in another. A healthy mucosa will not be so readily irritated

by either indigestible or partly decomposed food.

Osteopathic experimental work reveals that the vertebral and rib

lesions readily affect both the spinal nerves and the sympathetic

ganglia, which is followed by vasomotor and trophic disorder to the

mucous and submucous coats of the stomach, as shown by eccymosis and

hemorrhage of the submucosa and beginning parenchymatous degeneration

of the free ends of the glands of the mucosa. Upon the other hand

irritation of the muscles from dietetic errors always causes more or

less contraction of the muscles in the upper and middle dorsal, which,

in turn, may produce through imbalance of tension and fibrositic

changes, constant interosseous lesions and thus be the cause of the

catarrh becoming chronic. This vicious cycle phenomenon should not be

overlooked. Viscero-motor, viscerosensory and viscerotrophic reflexes

may be factors in the pathogenesis of the osteopathic lesion.

=Pathologically=, the mucous membrane is more or less covered with

mucus. Upon removal of the mucus the membrane is found red and swollen,

and the epithelial cells of the glands are granular. This is especially

noted in the pyloric area. There are minute extravasations of blood and

hemorrhages of the mucous coat, and infiltration of the submucous layer.

=Symptoms.=—Acute gastritis occurs at all ages, so particularly in

children care has to be taken that the attack is not the beginning

of some infectious disease. A careful inquiry into the history, and

examination of the vomitus will usually make the diagnosis clear.

The sudden onset of nausea, vomiting, pain in the epigastric region

referred to the back and head, vertigo in some cases, if the infections

can be ruled out should leave no doubt as to the nature of the disorder.

Other symptoms are weakness, and chilliness which later if the attack

is severe, is followed by fever. The tongue is coated, the lips dry,

and there may be herpes. Belching of gas, constipation in some and

diarrhea in others, and dark colored urine are noticeable. There is

tenderness on palpation over both the stomach and splanchnic areas.

Examination of the stomach contents show deficient hydrochloric

acid, the presence of organic acids, bile and undigested food, and

considerable mucus.

=Diagnosis.=—In young children acute gastric indigestion is common,

though a casual gastritis is rare. In the former prostration,

vomiting, and undigested, greenish stools are noted. In some cases

there is no fever, while in others it may range from 102 to 105

degrees. In all cases care should be taken, as has been stated,

that the attack is not the beginning of some infectious disease.

Appendicitis, acute bowel constriction, pregnancy, uremia, meningitis,

gall-stone colic, and gastric crises of tabes dorsalis should be

differentiated. Most attacks of acute dyspepsia are over in twenty-four

hours. The prognosis depends upon eliminating the cause. The X-ray may

be of value in protracted cases.

=Treatment.=—If the case is seen early, emptying the stomach by induced

vomiting or the stomach tube is the first indication. If several hours

have elapsed and much of the stomach contents have passed into the

intestine, emptying the colon with an enema will commonly give quick

relief. Withhold all food for from twelve to twenty-four hours, or

longer if necessary. In some cases the sipping of hot water will be

beneficial, while in others pellets of ice in the mouth will give some

relief.

Whether or not there existed previous spinal lesions there will always

be found muscular tension and spinal rigidness during an attack of

acute gastritis. These should be corrected for immediate relief, but

what is of greater importance, if these acute lesions are not corrected

the patient’s recuperative forces are interfered with and recovery is

delayed. Then, also, these lesions tend to chronicity and predispose

to future attacks. Treatment should be given daily, or oftener if

special indications arise. Though the most common area that demands

attention is from the fourth to tenth dorsals, still the vagi nerves,

especially the right, should not be neglected. Lesions of the upper

three cervicals are the most frequent disturbances of the vagi.

=Vomiting= is a common and distressing symptom. Pathologically, it is

due to an antiperistaltic contraction of the stomach and a spasmodic

contraction of the diaphragm and the abdominal muscles. It is caused,

usually, by irritation of the vagus in the stomach, or in the pharynx

by irritation along the spine (particularly in the cervical and upper

dorsal regions), or to the sympathetic nerves or to various parts of

the body, or by direct influence of the brain. Relief can usually

be given by inhibition of the vagus in the occipital region or by

inhibition at the fourth or fifth dorsal vertebra on the right side.

In a few instances, placing the patient in the knee-chest position and

gently raising the abdominal organs gives relief. If this does not

suffice the stomach and colon should be emptied, providing the vomiting

is protracted. A frequently effective measure for nausea and vomiting

that can be carried out by the attendant, is the application of hot

fomentations to the dorsal spine.

=Flatulency= may be very distressing. The spinal treatment may be

sufficient to control this condition, or careful direct pressure for a

few minutes over the pit of the stomach. Adjustment of the lower ribs,

especially of the left side, may be effective. Occasionally the gas

can be passed into the intestines by careful inhibitory treatment in

the region of the eighth and ninth dorsals. The inhibitory treatment

causes relaxation of the pyloric orifice; also, inhibition of the left

vagus relaxes the pylorus. Inhibition at the sixth and seventh dorsals

relaxes the cardiac orifice, thus favoring the passing of the gas from

the stomach out through the esophagus.

In all cases subject to gastritis the dorsal spine should receive

considerable attention in order that recovery may be complete. The

habits of the patient should be thoroughly regulated and overfatigue

guarded against. And, also of special importance in recurring attacks,

is the fact that a number of cases present some derangement of the

biliary tract, or duodenum, or the appendix region.

=Diet.=—After twenty-four or forty-eight hours, if the attack has been

severe, albumin water may be given in small quantities; also whey,

milk, bouillon, and chicken or lamb broth. If there is no return of

gastric distress, add junket, custard, cornstarch pudding, gelatine,

dropped eggs, scraped beef, and white meat of chicken; vegetables

purees made with cream or meat stock are usually well borne at this

time. Foods containing much cellulose, fats and sweets should be

withheld until all symptoms have subsided.

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