Nerve Deafness

The term “nerve deafness” is generally used very carelessly to apply to

any chronic or non-suppurative process of the labyrinthine structures

other than those mentioned above, which cause impaired hearing.

Nerve deafness is not an uncommon disease. In my cases of deafness

there has been some involvement of the labyrinthine structures or

auditory nerve in 27% of the cases examined. I think the reason for

most authors putting the percentage of nerve deafness much lower than

this is because of inexact methods of diagnosis. The above percentage

is based upon the actual measurement of nerve force. See measurement of

nerve force under non-suppurative otitis media above.

A careful study of cases by the method of actual measurement of nerve

force, shows that there are two distinct forms of nerve deafness.

In one there is only a deficient function of the structures of the

labyrinth, due perhaps to some perverted physiologic function, and this

form we may call auditory nerve deficiency. The other form of nerve

deafness, due probably to an actual degeneration of the nerve or its

end organs in the labyrinth, may be properly known as auditory nerve

degeneration.

=Auditory Nerve Deficiency.=—A study of our case reports shows that in

64% of the cases in which the nerve force was 16-30 or higher (more

than half) favorable results were obtained, provided that there was

no complicating labyrinthine affection. These cases have been classed

as “nerve deficiency” and the pathology as functional. A favorable

prognosis (64%) may be offered.

Example of tuning fork findings:

Fork 32 64 128 512 2048 4096

B. C. 18 20 20 20 10

A. C. T 50 60 60 40 20

In addition to the tuning fork findings the voice and watch test will

be reduced to from one-tenth to two-thirds normal. The patient often

complains of itching meati and dry nares. There are usually no signs or

symptoms of labyrinthine affection.

=Treatment.=—The treatment consists of local treatment to the

nasopharynx, tubes and meati as described under the treatment of

chronic otitis media. Everything should be done to build up the

patient’s general health and improve the local nutrition. It is highly

essential to search the entire system for sources of focal and general

infection. Auto-intoxication from chronic gastro-intestinal disease

was found in 90% of our cases. Any treatment therefore that will

restore normal gastro-intestinal function is indicated. Recently we

have had some excellent results from colonic irrigation and the proper

adjustment of diet in such cases. Any source of focal infection must of

course receive proper attention.

The osteopathic corrective treatment consists largely of careful

attention to lesions of the splanchnic area because of the importance

of normal digestion, metabolism and elimination. This is certainly a

most important part of the treatment and should never be neglected.

Upper cervical and mandibular lesions have much to do with the local

nutrition to the ear structures and these must not be neglected. The

fact that we almost constantly find evidence of deficient nutrition

to the meati and drums in this disease together with lesions of the

mandible, suggests a local osteopathic cause.

=Auditory Nerve Degeneration.=—In those cases in which there is a

measurable deficiency of nerve function of less than half the normal we

have found that very few respond to treatment. (See table above.) The

cause has therefore been attributed to a structural pathology and the

condition called auditory degeneration.

Example Table:

Fork 32 64 128 512 2048 4096

B. C. 10 12 8 5 2

A. C. S 20 25 15 7 5

There is usually very marked impairment of hearing for voice and all

other sounds. The Ingersoll watch may be heard five or ten inches, but

usually not at all, and the whispered voice heard only a few inches or

not at all. There are nearly always signs and symptoms of labyrinthine

deafness and evidence of tone islands. The deafness in these cases is

usually progressive regardless of any treatment.

In this disease there is nearly always an associated affection of the

labyrinth as shown by the high forks. The fractions represented by the

high forks will agree in proportion provided there is no labyrinthine

involvement.

Our results in auditory nerve degeneration have been measurable

improvement in only 2% of the cases treated. The prognosis is therefore

very poor and I believe we should always tell our patients frankly

that there is almost no chance for improving their hearing in such

cases. The treatment is the same as that given for nerve deficiency and

because of the general good that may be had from treatment, that is,

the improvement of the general resistance, it is often well for the

patient to have such treatment to stop the progress of further special

sensory degeneration.

It should always be our purpose to treat the patient rather than to

treat some particular organ only and if this method is followed, our

general results will surely be much higher.

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