ALOPECIA. POST PARTEM. ALOPECIA AREATA.

Normal shedding. Shedding out of time; laminitis, dropsy, exudative

dermatitis, acariasis, ringworm, traumas, folliculitis. Debility,

excessive lactation, starvation, petechial fever, spoiled fodder.

Without apparent cause, alopecia areata, neurosis, micrococci, disease

of derma. Symptoms: general disorder has general shedding. Local

disorder extends from a centre. In horse with increased pigmentation.

Treatment: correct general causes, use hair stimulants, cantharides,

kerosene, tar, pilocarpin, mercuric chloride, cresol, iodine, balsam

of Peru, silver nitrate. Arsenic.

Acquired baldness is recorded in horse, cattle, sheep and dog.

_Causes._ The simple shedding of hair occurs physiologically in animals

with the change of season, and if anything interferes with the growth of

the new hair a transient baldness may ensue. If such shedding occurs

from any cause at the wrong season, before the new hair has started, the

baldness may be accentuated. Thus shedding may occur in some forms of

indigestion, in laminitis, in dropsical swelling of the limbs or ventral

aspect of the body, in dermatitis with an exudation which concretes

around the hairs and raises them out of their follicles, in mange, in

demodectic acariasis, in circinate ringworm, in traumas as on the elbows

of dogs, etc., from lying on them, and in inflammation of the hair

follicles from a variety of infections. It has been charged on general

debility in excessive lactation, in gestation, and in starvation, on

poisons in the blood as in petechial fever, and on musty or spoiled

fodders in bad seasons, or from low damp lands.

When in the absence of such appreciable causes it commences at one or

more points and gradually extends, and persists, it constitutes alopecia

areata. This has been attributed to a disorder of the cutaneous nerves

(tropho-neurosis), but the progressive advance of the disease, without

limitation to areas representing the distribution of given cutaneous

nerves, and the complete absence of other derangement of nerve function,

throw doubt on this conclusion. Another doctrine attributes it to a

microbe, but though micrococci and other organisms have been found, they

have not been proved to be constant nor to be absolutely causative of

the disease. Still another theory holds that it is a disease of the

derma and not of the hair at all, the evulsion of the hair following the

implication of the tissues around the follicles.

_Symptoms._ The baldness dependent on a general disorder occurs at once

over an extended area. That of ringworm, acariasis, and of the specific

alopecia areata, advances gradually and often slowly from a given point,

until it may include a large area. Röll has seen it extend from a few

points to nearly the whole body of the horse in a single year. In this,

as in other cases in horse and dog, the baldness was followed by a

considerable increase of the pigmentation of the skin.

_Treatment._ In cases that occur as the result of other diseases, the

rational treatment is to deal with these diseases, and then to stimulate

the growth of hair by some one or other of the known stimulants (dilute

tincture of cantharides, kerosene, tar water, solution of pilocarpin

hydrochlorate). In the more specific form, no treatment has been very

successful, yet the best results on the whole appear to have come from

local germicide applications. Mercuric chloride in alcohol and water (1

∶ 500); cresol 1, alcohol 20; tincture of iodine reduced to half its

strength by addition of alcohol; balsam of Peru 1, alcohol 5; nitrate of

silver 1, alcohol 15, serve as examples.

As general treatment arsenic has been employed, but with no very

encouraging results.

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