TOXIC GASTRITIS IN SOLIPEDS.

Causes: caustics or irritants acting on mucosa, accidently, or

maliciously. Symptoms: colics, pinched face, small rapid pulse,

hurried breathing, hyperthermia, sometimes salivation, color of buccal

mucosa, odor, congestion of tongue, thirst, urination, icterus,

albuminuria, analysis of urine or vomited matter. Lesions: congestion,

corrosion, necrosis or ulceration of gastric mucosa, discoloration.

Treatment: antidote, stomach pump, demulcents, coagulants.

_Causes._ Toxic gastritis in solipeds is peculiar in this that it must

be due to one or other of the more caustic or irritant agents, which act

chemically on the tissues, while those agents that require to be

absorbed to establish a physiological irritation are comparatively

harmless. This depends on the fact that few or none of the poisonous

agents are absorbed by the gastric mucosa of the soliped, and if

ingested they must pass on into the duodenum before they can be absorbed

into the tissues and blood-vessels. Hence the horse is injured mainly by

actual caustics like mercuric chloride, zinc chloride, ferric or cupric

sulphate, the caustic alkalies or earths, or alkaline carbonates, and

the mineral acids. These may be taken accidently or administered

maliciously, or as medicines.

_Symptoms._ Morbid symptoms vary according to the agent swallowed. There

are colics, anxious countenance, small accelerated pulse, rapid

breathing, hyperthermia, and salivation, especially marked with mercuric

chloride. The buccal mucosa may give valuable indications, becoming

white with muriatic acid, or zinc, or antimony, or mercuric chloride,

yellow with nitric acid, and white changing to black with sulphuric acid

or silver nitrate. Ferric or cupric sulphate may give their respective

colors to the saliva, and the former will darken the fæces.

The mouth is usually dry, hot, and clammy, and the edges of the tongue

red. Temperature is usually elevated, yet with tartar emetic it may be

distinctly reduced. Thirst is usually marked, and urination frequent.

Icterus and albuminuria attend on phosphorus poisoning. When vomiting

takes place the appearance or analysis of the matters rejected, or

otherwise of the urine, will often indicate the nature of the poison.

_Lesions._ The gastric mucosa is congested and discolored, but the

corrosion and even the ulceration are especially characteristic. Patches

of necrotic mucous membrane may be more or less detached exposing a deep

red submucosa. The coloration otherwise varies;—white or black with

sulphuric acid or silver nitrate; white with muriatic acid, the caustic

alkalies, or zinc chloride; yellow with nitric acid; or green with salts

of copper.

Similar lesions are found on the buccal, œsophagean and intestinal

mucosæ, and even at times on the respiratory.

_Treatment._ In the treatment of this form of gastritis the first

consideration is to expel, or use an antidote to, the poison. In the

soliped, emetics are useless. The stomach pump or tube may, however, be

applied with good effect in nearly all cases, alternately throwing in

water and drawing it off. Demulcents and coagulants are also universally

applicable. Milk, eggs beaten up in milk, blood albumen, flaxseed tea,

well boiled gruels, or slippery elm bark, may be used as may be most

convenient. Next come the other chemical antidotes the use of which

however demands a previous knowledge of the poison present. For the

mineral acids one can make use of calcined magnesia, lime water, chalk,

or carbonate of soda in weak solution. For alkalies the appropriate

antidote is vinegar. For carbolic acid, vinegar, alcohol, or failing

these a weak solution of soda or oil. For tartar emetic, gallic or

tannic acid. For bichromate of potash or chromic acid, calcined

magnesia, magnesia carbonate, or lime carbonate. For phosphorus, old oil

of turpentine and demulcents—no oil. For ammonia, vinegar followed by

almond, olive or sweet oil. In case of œdema glottidis, tracheotomy. For

copper salts yellow prussiate of potash, which precipitates the copper

in an insoluble form, and demulcents. For mercuric chloride, demulcent

drinks can be resorted to, there is no other reliable antidote. In all

cases after the evacuation of the stomach and the use of the antidote,

mucilaginous agents must be given freely with morphia or other anodynes.

Text book of veterinary medicine, Volume 2 (of 5) · 第 83 / 308 题